Guardat en:
| Autor principal: | |
|---|---|
| Format: | Recurso digital |
| Idioma: | anglès |
| Publicat: |
Zenodo
2025
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| Accés en línia: | https://doi.org/10.5281/zenodo.17614585 |
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Taula de continguts:
- <p><strong><span>Jalodara</span></strong><strong><span> </span></strong><strong><span>(Ascites)</span></strong></p> <p><span>Udara<span> </span>Roga<span> </span>denotes<span> </span>the<span> </span>generalized<span> </span>distension<span> </span>or<span> </span>enlargement<span> </span>of<span> </span>abdomen<span> </span>of<span> </span>varied<span> </span>aetiology.<span> </span>It<span> </span>is one among the Ashtamahagada. From the origin of the illness it is difficult to manage. Agni dosha and mala vriddhi causes vitiation of Prana, Agni and Apana and obstruction of the upward and downward<span> </span>channels<span> </span>of<span> </span>circulation.<span> </span></span></p> <p><span> </span><span>The<span> </span>vitiated<span> </span>doshas<span> </span>get<span> </span>lodged<span> </span>between<span> </span>skin<span> </span>and<span> </span>muscle<span> </span>tissue and causes extensive distension of the abdomen resulting in Udara roga. The cardinal features are Kukshi adhmana (enlargement of abdomen), Karapada<span> </span>shopha<span> </span>(oedema<span> </span>in<span> </span>the<span> </span>limbs),<span> </span>Mandagni/<span> </span>Atyanta<span> </span>Nastagni,<span> </span>Krushagatra(emaciation Extreme<span> </span>impairment of<span> </span>agni (digestion<span> </span>and<span> </span>metabolism) is the basic pathology<span> </span>of<span> </span>udara roga in general,<span> </span>which<span> </span>is<span> </span>regarded<span> </span>as one<span> </span>among<span> </span>the<span> </span>mahagada<span> </span>(major<span> </span>illness).<span> </span>It<span> </span>is<span> </span>classified<span> </span>into<span> </span>eight<span> </span>types Vatodara (Accumulation of flatus), Pittodara (Hepatic causes), Kaphodara (Renal causes), <span>Sannipatodara</span><span> </span><span>(exudative </span>causes),<span> </span>Plihodara<span> </span>(splenomegaly),<span> </span>kshatodara/chhidrodara<span> </span>(abdominal<span> </span>enlargement<span> </span>due<span> </span>to intestinal perforation), baddhagudodara (enlargement of abdomen due to gastrointestinal obstruction)<span> </span>and<span> </span>jalodara/udakodara<span> </span>(ascites).<span> </span>Yakritodara<span> </span>(hepatomegaly)<span> </span>is<span> </span>also<span> </span>a<span> </span>distinct type<span> </span>of<span> </span>udara<span> </span>roga<span> </span>but<span> </span>incorporated<span> </span>in<span> </span>the<span> </span>plihodara<span> </span>since<span> </span>the<span> </span>aetiology<span> </span>and<span> </span>treatment<span> </span>of<span> </span>these<span> </span>two conditions are similar. The disease closely resembles Ascites, which is pathological accumulation of fluid within the peritoneal cavity. The mortality increases from complications such as spontaneous bacterial<span> </span>peritonitis<span> </span>and<span> </span>hepato-renal<span> </span>syndrome.<span> </span>Mortality ranges from<span> </span>15% in<span> </span>one<span> </span>year<span> </span>to<span> </span>44% in<span> </span>5 years. Treatment in contemporary science for Ascites<span> </span>include<span> </span>trans<span> </span>jugular<span> </span>intra<span> </span>hepatic<span> </span>portosystemic<span> </span>Shunt,<span> </span>Diuretics<span> </span>etc<span> </span>which<span> </span>only<span> </span>provide provisional relief with time dependent recurrence but fluid gets collected in peritoneal<span> </span>cavity<span> </span>repeatedly.<span> </span>In<span> </span>such<span> </span>case,<span> </span>Ayurvedic<span> </span>treatment<span> </span>gives<span> </span>relief<span> </span>without<span> </span>any<span> </span><span>sideffect.</span></span></p>