Gardado en:
| Autor Principal: | |
|---|---|
| Formato: | Recurso digital |
| Idioma: | |
| Publicado: |
Zenodo
2026
|
| Subjects: | |
| Acceso en liña: | https://doi.org/10.5281/zenodo.19785263 |
| Tags: |
Engadir etiqueta
Sen Etiquetas, Sexa o primeiro en etiquetar este rexistro!
|
Table of Contents:
- <p>This paper presents a mechanistic hypothesis for the phenomenon of trembling under psychological stress. Contrary to evolutionary psychology interpretations that implicitly treat motor output as direct behavioral expression, we propose that trembling arises from the failure of predictive neural coding in motor control circuits. Central to this model is the state-dependent effect of norepinephrine (NE) on signal-to-noise ratio (SNR): while NE amplifies neural gain indiscriminately, the functional outcome depends entirely on the stability of underlying attractor sequences. In systems with robust, well-established attractor basins, NE sharpens signal discrimination; in systems with shallow, competing attractors, NE causes noise to flood into adjacent basins, generating non-directed micro-corrections perceived as motor errors. We integrate this core mechanism with dopaminergic modulation (Drd1/Drd2 balance), glutamate receptor trafficking (GluR1/GluR2/3), and the opponent relationship between the ventral tegmental area (VTA) and lateral habenula (LHb). The model reframes trembling not as phylogenetic vestige but as real-time dynamical systems failure---specifically, the inability of selection mechanisms to resolve conflicts at the rate they are generated.</p>