Advancing Understanding of Long COVID Pathophysiology Through Quantum Walk-Based Network Analysis

Fuente: arXiv
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Autores principales: Park, Jaesub, Hwang, Woochang, Lee, Seokjun, Lee, Hyun Chang, MacMahon, Méabh, Zilbauer, Matthias, Han, Namshik
Formato: Preprint
Publicado: 2025
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author Park, Jaesub
Hwang, Woochang
Lee, Seokjun
Lee, Hyun Chang
MacMahon, Méabh
Zilbauer, Matthias
Han, Namshik
author_facet Park, Jaesub
Hwang, Woochang
Lee, Seokjun
Lee, Hyun Chang
MacMahon, Méabh
Zilbauer, Matthias
Han, Namshik
contents Long COVID is a multisystem condition characterized by persistent symptoms such as fatigue, cognitive impairment, and systemic inflammation, following COVID-19 infection, yet its mechanisms remain poorly understood. In this study, we applied quantum walk (QW), a computational approach leveraging quantum interference, to explore large-scale SARS-CoV-2-induced protein (SIP) networks. Compared to the conventional random walk with restart (RWR) method, QW demonstrated superior capacity to traverse deeper regions of the network, uncovering proteins and pathways implicated in Long COVID. Key findings include mitochondrial dysfunction, thromboinflammatory responses, and neuronal inflammation as central mechanisms. QW uniquely identified the CDGSH iron-sulfur domain-containing protein family and VDAC1, a mitochondrial calcium transporter, as critical regulators of these processes. VDAC1 emerged as a potential biomarker and therapeutic target, supported by FDA-approved compounds such as cannabidiol. These findings highlight QW as a powerful tool for elucidating complex biological systems and identifying novel therapeutic targets for conditions like Long COVID.
format Preprint
id arxiv_https___arxiv_org_abs_2501_15208
institution arXiv
publishDate 2025
record_format arxiv
spellingShingle Advancing Understanding of Long COVID Pathophysiology Through Quantum Walk-Based Network Analysis
Park, Jaesub
Hwang, Woochang
Lee, Seokjun
Lee, Hyun Chang
MacMahon, Méabh
Zilbauer, Matthias
Han, Namshik
Molecular Networks
Long COVID is a multisystem condition characterized by persistent symptoms such as fatigue, cognitive impairment, and systemic inflammation, following COVID-19 infection, yet its mechanisms remain poorly understood. In this study, we applied quantum walk (QW), a computational approach leveraging quantum interference, to explore large-scale SARS-CoV-2-induced protein (SIP) networks. Compared to the conventional random walk with restart (RWR) method, QW demonstrated superior capacity to traverse deeper regions of the network, uncovering proteins and pathways implicated in Long COVID. Key findings include mitochondrial dysfunction, thromboinflammatory responses, and neuronal inflammation as central mechanisms. QW uniquely identified the CDGSH iron-sulfur domain-containing protein family and VDAC1, a mitochondrial calcium transporter, as critical regulators of these processes. VDAC1 emerged as a potential biomarker and therapeutic target, supported by FDA-approved compounds such as cannabidiol. These findings highlight QW as a powerful tool for elucidating complex biological systems and identifying novel therapeutic targets for conditions like Long COVID.
title Advancing Understanding of Long COVID Pathophysiology Through Quantum Walk-Based Network Analysis
topic Molecular Networks
url https://arxiv.org/abs/2501.15208