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Main Authors: Li, Jiarui, Yin, Zixiang, Smith, Haley, Ding, Zhengming, Landry, Samuel J., Mettu, Ramgopal R.
Format: Preprint
Published: 2025
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Online Access:https://arxiv.org/abs/2507.03197
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author Li, Jiarui
Yin, Zixiang
Smith, Haley
Ding, Zhengming
Landry, Samuel J.
Mettu, Ramgopal R.
author_facet Li, Jiarui
Yin, Zixiang
Smith, Haley
Ding, Zhengming
Landry, Samuel J.
Mettu, Ramgopal R.
contents CD8+ "killer" T cells and CD4+ "helper" T cells play a central role in the adaptive immune system by recognizing antigens presented by Major Histocompatibility Complex (pMHC) molecules via T Cell Receptors (TCRs). Modeling binding between T cells and the pMHC complex is fundamental to understanding basic mechanisms of human immune response as well as in developing therapies. While transformer-based models such as TULIP have achieved impressive performance in this domain, their black-box nature precludes interpretability and thus limits a deeper mechanistic understanding of T cell response. Most existing post-hoc explainable AI (XAI) methods are confined to encoder-only, co-attention, or model-specific architectures and cannot handle encoder-decoder transformers used in TCR-pMHC modeling. To address this gap, we propose Quantifying Cross-Attention Interaction (QCAI), a new post-hoc method designed to interpret the cross-attention mechanisms in transformer decoders. Quantitative evaluation is a challenge for XAI methods; we have compiled TCR-XAI, a benchmark consisting of 274 experimentally determined TCR-pMHC structures to serve as ground truth for binding. Using these structures we compute physical distances between relevant amino acid residues in the TCR-pMHC interaction region and evaluate how well our method and others estimate the importance of residues in this region across the dataset. We show that QCAI achieves state-of-the-art performance on both interpretability and prediction accuracy under the TCR-XAI benchmark.
format Preprint
id arxiv_https___arxiv_org_abs_2507_03197
institution arXiv
publishDate 2025
record_format arxiv
spellingShingle Quantifying Cross-Attention Interaction in Transformers for Interpreting TCR-pMHC Binding
Li, Jiarui
Yin, Zixiang
Smith, Haley
Ding, Zhengming
Landry, Samuel J.
Mettu, Ramgopal R.
Computational Engineering, Finance, and Science
Machine Learning
Biomolecules
CD8+ "killer" T cells and CD4+ "helper" T cells play a central role in the adaptive immune system by recognizing antigens presented by Major Histocompatibility Complex (pMHC) molecules via T Cell Receptors (TCRs). Modeling binding between T cells and the pMHC complex is fundamental to understanding basic mechanisms of human immune response as well as in developing therapies. While transformer-based models such as TULIP have achieved impressive performance in this domain, their black-box nature precludes interpretability and thus limits a deeper mechanistic understanding of T cell response. Most existing post-hoc explainable AI (XAI) methods are confined to encoder-only, co-attention, or model-specific architectures and cannot handle encoder-decoder transformers used in TCR-pMHC modeling. To address this gap, we propose Quantifying Cross-Attention Interaction (QCAI), a new post-hoc method designed to interpret the cross-attention mechanisms in transformer decoders. Quantitative evaluation is a challenge for XAI methods; we have compiled TCR-XAI, a benchmark consisting of 274 experimentally determined TCR-pMHC structures to serve as ground truth for binding. Using these structures we compute physical distances between relevant amino acid residues in the TCR-pMHC interaction region and evaluate how well our method and others estimate the importance of residues in this region across the dataset. We show that QCAI achieves state-of-the-art performance on both interpretability and prediction accuracy under the TCR-XAI benchmark.
title Quantifying Cross-Attention Interaction in Transformers for Interpreting TCR-pMHC Binding
topic Computational Engineering, Finance, and Science
Machine Learning
Biomolecules
url https://arxiv.org/abs/2507.03197