White spot syndrome virus IE1 protein blocks the integrin-FAK signaling to enhance viral infection in shrimp.

Fuente: PubMed
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Main Authors: Lu, Kaiyu, Chen, Xiyu, Zhang, Huimin, Zhu, Jinghua, Zhao, Yongzhen, Chen, Xiuli, Zhang, Yueling, Yao, Defu
Format: Artículo científico
Language:en
Published: Fish & shellfish immunology 2025
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author Lu, Kaiyu
Chen, Xiyu
Zhang, Huimin
Zhu, Jinghua
Zhao, Yongzhen
Chen, Xiuli
Zhang, Yueling
Yao, Defu
author_facet Lu, Kaiyu
Chen, Xiyu
Zhang, Huimin
Zhu, Jinghua
Zhao, Yongzhen
Chen, Xiuli
Zhang, Yueling
Yao, Defu
Lu, Kaiyu
Chen, Xiyu
Zhang, Huimin
Zhu, Jinghua
Zhao, Yongzhen
Chen, Xiuli
Zhang, Yueling
Yao, Defu
collection PubMed - marine biology
contents White spot syndrome virus IE1 protein blocks the integrin-FAK signaling to enhance viral infection in shrimp. Lu, Kaiyu Chen, Xiyu Zhang, Huimin Zhu, Jinghua Zhao, Yongzhen Chen, Xiuli Zhang, Yueling Yao, Defu Animals Penaeidae White spot syndrome virus 1 Signal Transduction Arthropod Proteins Integrins Cell Adhesion DNA viruses commonly utilize immediate-early proteins to manipulate cellular signaling pathways in order to facilitate their infection. Our previous research has suggested that IE1, an immediate-early protein encoded by the white spot syndrome virus (WSSV), may modulate the shrimp integrin-FAK signaling pathway. However, the specific molecular mechanism and role of IE1 in regulating this signaling pathway remain unclear. In this study, we demonstrated that IE1 competes for binding to the cytoplasmic tail of Penaeus vannamei integrin-α5, resulting in the inhibition of the integrin-α5-FAK interaction, thereby suppressing FAK activation and cell adhesion. Furthermore, we observed a significant increase in the expression of P. vannamei integrin-α5 and FAK following WSSV infection. Additionally, knockdown of integrin-α5 or FAK through RNA interference has been shown to reduce cell adhesion and enhance WSSV infection. In conclusion, our findings reveal that IE1 disrupts integrin-FAK signaling to inhibit cell adhesion, ultimately promoting WSSV infection in shrimp.
format Artículo científico
id pubmed_39637952
institution PubMed
language en
publishDate 2025
publisher Fish & shellfish immunology
record_format pubmed
spellingShingle White spot syndrome virus IE1 protein blocks the integrin-FAK signaling to enhance viral infection in shrimp.
Lu, Kaiyu
Chen, Xiyu
Zhang, Huimin
Zhu, Jinghua
Zhao, Yongzhen
Chen, Xiuli
Zhang, Yueling
Yao, Defu
Animals
Penaeidae
White spot syndrome virus 1
Signal Transduction
Arthropod Proteins
Integrins
Cell Adhesion
White spot syndrome virus IE1 protein blocks the integrin-FAK signaling to enhance viral infection in shrimp. Lu, Kaiyu Chen, Xiyu Zhang, Huimin Zhu, Jinghua Zhao, Yongzhen Chen, Xiuli Zhang, Yueling Yao, Defu Animals Penaeidae White spot syndrome virus 1 Signal Transduction Arthropod Proteins Integrins Cell Adhesion DNA viruses commonly utilize immediate-early proteins to manipulate cellular signaling pathways in order to facilitate their infection. Our previous research has suggested that IE1, an immediate-early protein encoded by the white spot syndrome virus (WSSV), may modulate the shrimp integrin-FAK signaling pathway. However, the specific molecular mechanism and role of IE1 in regulating this signaling pathway remain unclear. In this study, we demonstrated that IE1 competes for binding to the cytoplasmic tail of Penaeus vannamei integrin-α5, resulting in the inhibition of the integrin-α5-FAK interaction, thereby suppressing FAK activation and cell adhesion. Furthermore, we observed a significant increase in the expression of P. vannamei integrin-α5 and FAK following WSSV infection. Additionally, knockdown of integrin-α5 or FAK through RNA interference has been shown to reduce cell adhesion and enhance WSSV infection. In conclusion, our findings reveal that IE1 disrupts integrin-FAK signaling to inhibit cell adhesion, ultimately promoting WSSV infection in shrimp.
title White spot syndrome virus IE1 protein blocks the integrin-FAK signaling to enhance viral infection in shrimp.
topic Animals
Penaeidae
White spot syndrome virus 1
Signal Transduction
Arthropod Proteins
Integrins
Cell Adhesion
url https://pubmed.ncbi.nlm.nih.gov/39637952/