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Main Authors: Nie, Li, Wu, Xiang-Yu, Zhao, Zi-Yue, Fei, Chen-Jie, Zhu, Ting-Fang, Shao, Jian-Zhong, Chen, Jiong
Format: Artículo científico
Language:en
Published: Zoological research 2025
Subjects:
Online Access:https://pubmed.ncbi.nlm.nih.gov/39757016/
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author Nie, Li
Wu, Xiang-Yu
Zhao, Zi-Yue
Fei, Chen-Jie
Zhu, Ting-Fang
Shao, Jian-Zhong
Chen, Jiong
author_facet Nie, Li
Wu, Xiang-Yu
Zhao, Zi-Yue
Fei, Chen-Jie
Zhu, Ting-Fang
Shao, Jian-Zhong
Chen, Jiong
Nie, Li
Wu, Xiang-Yu
Zhao, Zi-Yue
Fei, Chen-Jie
Zhu, Ting-Fang
Shao, Jian-Zhong
Chen, Jiong
collection PubMed - marine biology
contents Palmitoylation-mediated NLRP3 inflammasome activation in teleosts highlights evolutionary divergence in immune regulation. Nie, Li Wu, Xiang-Yu Zhao, Zi-Yue Fei, Chen-Jie Zhu, Ting-Fang Shao, Jian-Zhong Chen, Jiong Animals NLR Family, Pyrin Domain-Containing 3 Protein Inflammasomes Lipoylation Zebrafish Biological Evolution Immunity, Innate Perciformes NLRP3 inflammasome activation is pivotal for cytokine secretion and pyroptosis in response to diverse stimuli, playing a crucial role in innate immunity. While extensively studied in mammals, the regulatory mechanisms governing NLRP3 activation in non-mammalian vertebrates remain largely unexplored. Teleosts, as basal vertebrates, represent an ideal model for exploring the evolutionary trajectory of inflammasome regulation. In this study, ABE assays, confocal microscopy, and biochemical analyses were applied to systematically characterize the mechanisms underlying NLRP3 inflammasome in teleosts, using large yellow croakers ( , ) and zebrafish ( , ) as representative models. Our findings revealed a previously unrecognized palmitoylation-dependent regulatory mechanism essential for teleost NLRP3 activation. Specifically, zDHHC18-mediated palmitoylation at a teleost-specific cysteine residue (C946 in NLRP3, C1037 in NLRP3) was required for the translocation of NLRP3 to the dispersed trans-Golgi network, facilitating its subsequent recruitment to the microtubule-organizing center. This membrane trafficking was crucial for inflammasome assembly and downstream inflammatory responses. These findings provide new insights into the distinct regulatory mechanisms of NLRP3 activation in teleosts, highlighting an evolutionary divergence that contributes to innate immunity adaptation in early vertebrates.
format Artículo científico
id pubmed_39757016
institution PubMed
language en
publishDate 2025
publisher Zoological research
record_format pubmed
spellingShingle Palmitoylation-mediated NLRP3 inflammasome activation in teleosts highlights evolutionary divergence in immune regulation.
Nie, Li
Wu, Xiang-Yu
Zhao, Zi-Yue
Fei, Chen-Jie
Zhu, Ting-Fang
Shao, Jian-Zhong
Chen, Jiong
Animals
NLR Family, Pyrin Domain-Containing 3 Protein
Inflammasomes
Lipoylation
Zebrafish
Biological Evolution
Immunity, Innate
Perciformes
Palmitoylation-mediated NLRP3 inflammasome activation in teleosts highlights evolutionary divergence in immune regulation. Nie, Li Wu, Xiang-Yu Zhao, Zi-Yue Fei, Chen-Jie Zhu, Ting-Fang Shao, Jian-Zhong Chen, Jiong Animals NLR Family, Pyrin Domain-Containing 3 Protein Inflammasomes Lipoylation Zebrafish Biological Evolution Immunity, Innate Perciformes NLRP3 inflammasome activation is pivotal for cytokine secretion and pyroptosis in response to diverse stimuli, playing a crucial role in innate immunity. While extensively studied in mammals, the regulatory mechanisms governing NLRP3 activation in non-mammalian vertebrates remain largely unexplored. Teleosts, as basal vertebrates, represent an ideal model for exploring the evolutionary trajectory of inflammasome regulation. In this study, ABE assays, confocal microscopy, and biochemical analyses were applied to systematically characterize the mechanisms underlying NLRP3 inflammasome in teleosts, using large yellow croakers ( , ) and zebrafish ( , ) as representative models. Our findings revealed a previously unrecognized palmitoylation-dependent regulatory mechanism essential for teleost NLRP3 activation. Specifically, zDHHC18-mediated palmitoylation at a teleost-specific cysteine residue (C946 in NLRP3, C1037 in NLRP3) was required for the translocation of NLRP3 to the dispersed trans-Golgi network, facilitating its subsequent recruitment to the microtubule-organizing center. This membrane trafficking was crucial for inflammasome assembly and downstream inflammatory responses. These findings provide new insights into the distinct regulatory mechanisms of NLRP3 activation in teleosts, highlighting an evolutionary divergence that contributes to innate immunity adaptation in early vertebrates.
title Palmitoylation-mediated NLRP3 inflammasome activation in teleosts highlights evolutionary divergence in immune regulation.
topic Animals
NLR Family, Pyrin Domain-Containing 3 Protein
Inflammasomes
Lipoylation
Zebrafish
Biological Evolution
Immunity, Innate
Perciformes
url https://pubmed.ncbi.nlm.nih.gov/39757016/