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Autori principali: Wan, Ying, Rong, Lu, Li, Qingyuan, Liu, Suzhi, Li, Wentao, Ye, Min, Luo, Weifeng, Xie, Anmu, Shao, Jingsong, Guo, Dengjun, Zhang, Xiaoping, Zhang, Kezhong, Liu, Zhenguo
Natura: Artículo científico
Lingua:en
Pubblicazione: Molecular neurobiology 2025
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Accesso online:https://pubmed.ncbi.nlm.nih.gov/40299300/
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author Wan, Ying
Rong, Lu
Li, Qingyuan
Liu, Suzhi
Li, Wentao
Ye, Min
Luo, Weifeng
Xie, Anmu
Shao, Jingsong
Guo, Dengjun
Zhang, Xiaoping
Zhang, Kezhong
Liu, Zhenguo
author_facet Wan, Ying
Rong, Lu
Li, Qingyuan
Liu, Suzhi
Li, Wentao
Ye, Min
Luo, Weifeng
Xie, Anmu
Shao, Jingsong
Guo, Dengjun
Zhang, Xiaoping
Zhang, Kezhong
Liu, Zhenguo
Wan, Ying
Rong, Lu
Li, Qingyuan
Liu, Suzhi
Li, Wentao
Ye, Min
Luo, Weifeng
Xie, Anmu
Shao, Jingsong
Guo, Dengjun
Zhang, Xiaoping
Zhang, Kezhong
Liu, Zhenguo
collection PubMed - marine biology
contents Integrative Approaches Identify Genetic Determinants of Levodopa Induced Dyskinesia. Wan, Ying Rong, Lu Li, Qingyuan Liu, Suzhi Li, Wentao Ye, Min Luo, Weifeng Xie, Anmu Shao, Jingsong Guo, Dengjun Zhang, Xiaoping Zhang, Kezhong Liu, Zhenguo Humans Levodopa Genome-Wide Association Study Dyskinesia, Drug-Induced Genetic Predisposition to Disease Male Female Polymorphism, Single Nucleotide Quantitative Trait Loci Parkinson Disease Middle Aged Aged Prospective Studies Levodopa induced dyskinesia (LID) is a serious side effect of levodopa treatment in Parkinson's disease (PD), with limited interventions. Understanding the genetic impact on LID would help inform future intervention studies. We performed integrative genomic analysis approaches to identify the genetic determinants of LID in a Chinese multi-center prospective, observational PD cohort. In this cohort, 46 of 315 PD patients developed LID during 2.5 years of follow-up. First, we performed a genome-wide association study (GWAS) in this cohort, followed by a meta-analysis integrating our GWAS summary data with additional data of European ancestry. Both GWAS analyses identified the Bromodomain Containing 3 (BRD3) as a LID susceptibility gene (P
format Artículo científico
id pubmed_40299300
institution PubMed
language en
publishDate 2025
publisher Molecular neurobiology
record_format pubmed
spellingShingle Integrative Approaches Identify Genetic Determinants of Levodopa Induced Dyskinesia.
Wan, Ying
Rong, Lu
Li, Qingyuan
Liu, Suzhi
Li, Wentao
Ye, Min
Luo, Weifeng
Xie, Anmu
Shao, Jingsong
Guo, Dengjun
Zhang, Xiaoping
Zhang, Kezhong
Liu, Zhenguo
Humans
Levodopa
Genome-Wide Association Study
Dyskinesia, Drug-Induced
Genetic Predisposition to Disease
Male
Female
Polymorphism, Single Nucleotide
Quantitative Trait Loci
Parkinson Disease
Middle Aged
Aged
Prospective Studies
Integrative Approaches Identify Genetic Determinants of Levodopa Induced Dyskinesia. Wan, Ying Rong, Lu Li, Qingyuan Liu, Suzhi Li, Wentao Ye, Min Luo, Weifeng Xie, Anmu Shao, Jingsong Guo, Dengjun Zhang, Xiaoping Zhang, Kezhong Liu, Zhenguo Humans Levodopa Genome-Wide Association Study Dyskinesia, Drug-Induced Genetic Predisposition to Disease Male Female Polymorphism, Single Nucleotide Quantitative Trait Loci Parkinson Disease Middle Aged Aged Prospective Studies Levodopa induced dyskinesia (LID) is a serious side effect of levodopa treatment in Parkinson's disease (PD), with limited interventions. Understanding the genetic impact on LID would help inform future intervention studies. We performed integrative genomic analysis approaches to identify the genetic determinants of LID in a Chinese multi-center prospective, observational PD cohort. In this cohort, 46 of 315 PD patients developed LID during 2.5 years of follow-up. First, we performed a genome-wide association study (GWAS) in this cohort, followed by a meta-analysis integrating our GWAS summary data with additional data of European ancestry. Both GWAS analyses identified the Bromodomain Containing 3 (BRD3) as a LID susceptibility gene (P
title Integrative Approaches Identify Genetic Determinants of Levodopa Induced Dyskinesia.
topic Humans
Levodopa
Genome-Wide Association Study
Dyskinesia, Drug-Induced
Genetic Predisposition to Disease
Male
Female
Polymorphism, Single Nucleotide
Quantitative Trait Loci
Parkinson Disease
Middle Aged
Aged
Prospective Studies
url https://pubmed.ncbi.nlm.nih.gov/40299300/