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Main Authors: Chen, Shanduo, Gildor, Tsvia, Tewari, Prashant, Ben-Tabou de-Leon, Smadar
Format: Artículo científico
Language:en
Published: Journal of experimental zoology. Part B, Molecular and developmental evolution 2026
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Online Access:https://pubmed.ncbi.nlm.nih.gov/40855725/
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author Chen, Shanduo
Gildor, Tsvia
Tewari, Prashant
Ben-Tabou de-Leon, Smadar
author_facet Chen, Shanduo
Gildor, Tsvia
Tewari, Prashant
Ben-Tabou de-Leon, Smadar
Chen, Shanduo
Gildor, Tsvia
Tewari, Prashant
Ben-Tabou de-Leon, Smadar
collection PubMed - marine biology
contents Skeletogenic Expression of Integrin Alpha, Talin and Npnt Genes and Npnt Role in Sea Urchin Skeletogenesis. Chen, Shanduo Gildor, Tsvia Tewari, Prashant Ben-Tabou de-Leon, Smadar Animals Talin Gene Expression Regulation, Developmental Extracellular Matrix Proteins Paracentrotus Integrins Sea Urchins Biomineralization, the formation of mineralized tissues like skeletons and shells, is an essential developmental process in diverged phyla. Vertebrates' biomineralization involves the secretion of specialized extracellular matrix (ECM) proteins and the formation of Integrin-based focal adhesions, yet less is known about the role of such factors in invertebrates. A recent study has shown that focal adhesions form around the calcite spicule of the sea urchin larva, however, the skeletogenic expression and role of adhesion related proteins in this system are understudied. Here, we identified a set of ECM and adhesion genes that show enriched expression in the sea urchin skeletogenic cells and studied the role of the ECM protein, Npnt, in Paracentrotus lividus. The integrin alpha proteins, Pl-Ahi, Pl-Aji, Pl-Api, and the Pl-Talin protein are highly conserved between sea urchin and humans and the expression of these genes is enriched in the skeletogenic cells during early skeletogenesis. Pl-npnt is expressed specifically in skeletogenic cells throughout skeletogenesis and requires Vascular Endothelial Growth Factor (VEGF) signaling for its maintenance. Genetic perturbations of Pl-npnt result in skeletal defects, including reduced length of skeletal rods, ectopic spicule formation and branching, while skeletogenic cell migration remained unaffected. The activation of focal adhesion kinase (FAK) around the spicules is independent of Pl-Npnt activity in agreement with the loss of Integrin binding site in the sea urchin Npnt protein. Our findings set the stage for further analyses of ECM and adhesion-mediated mechanisms that drive sea urchin biomineralization, and most likely participate in skeletal development across metazoans.
format Artículo científico
id pubmed_40855725
institution PubMed
language en
publishDate 2026
publisher Journal of experimental zoology. Part B, Molecular and developmental evolution
record_format pubmed
spellingShingle Skeletogenic Expression of Integrin Alpha, Talin and Npnt Genes and Npnt Role in Sea Urchin Skeletogenesis.
Chen, Shanduo
Gildor, Tsvia
Tewari, Prashant
Ben-Tabou de-Leon, Smadar
Animals
Talin
Gene Expression Regulation, Developmental
Extracellular Matrix Proteins
Paracentrotus
Integrins
Sea Urchins
Skeletogenic Expression of Integrin Alpha, Talin and Npnt Genes and Npnt Role in Sea Urchin Skeletogenesis. Chen, Shanduo Gildor, Tsvia Tewari, Prashant Ben-Tabou de-Leon, Smadar Animals Talin Gene Expression Regulation, Developmental Extracellular Matrix Proteins Paracentrotus Integrins Sea Urchins Biomineralization, the formation of mineralized tissues like skeletons and shells, is an essential developmental process in diverged phyla. Vertebrates' biomineralization involves the secretion of specialized extracellular matrix (ECM) proteins and the formation of Integrin-based focal adhesions, yet less is known about the role of such factors in invertebrates. A recent study has shown that focal adhesions form around the calcite spicule of the sea urchin larva, however, the skeletogenic expression and role of adhesion related proteins in this system are understudied. Here, we identified a set of ECM and adhesion genes that show enriched expression in the sea urchin skeletogenic cells and studied the role of the ECM protein, Npnt, in Paracentrotus lividus. The integrin alpha proteins, Pl-Ahi, Pl-Aji, Pl-Api, and the Pl-Talin protein are highly conserved between sea urchin and humans and the expression of these genes is enriched in the skeletogenic cells during early skeletogenesis. Pl-npnt is expressed specifically in skeletogenic cells throughout skeletogenesis and requires Vascular Endothelial Growth Factor (VEGF) signaling for its maintenance. Genetic perturbations of Pl-npnt result in skeletal defects, including reduced length of skeletal rods, ectopic spicule formation and branching, while skeletogenic cell migration remained unaffected. The activation of focal adhesion kinase (FAK) around the spicules is independent of Pl-Npnt activity in agreement with the loss of Integrin binding site in the sea urchin Npnt protein. Our findings set the stage for further analyses of ECM and adhesion-mediated mechanisms that drive sea urchin biomineralization, and most likely participate in skeletal development across metazoans.
title Skeletogenic Expression of Integrin Alpha, Talin and Npnt Genes and Npnt Role in Sea Urchin Skeletogenesis.
topic Animals
Talin
Gene Expression Regulation, Developmental
Extracellular Matrix Proteins
Paracentrotus
Integrins
Sea Urchins
url https://pubmed.ncbi.nlm.nih.gov/40855725/