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Main Authors: Deng, Hongyan, Jia, Shuke, Zhu, Chunchun, Hua, Jiale, Wang, Zixuan, Sun, Xueyi, Liu, Wen, Shi, Liyun, Li, Wenhua, Gui, Jian-Fang, Liu, Xing, Xiao, Wuhan
Format: Artículo científico
Language:en
Published: Cell reports 2026
Subjects:
Online Access:https://pubmed.ncbi.nlm.nih.gov/41520336/
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author Deng, Hongyan
Jia, Shuke
Zhu, Chunchun
Hua, Jiale
Wang, Zixuan
Sun, Xueyi
Liu, Wen
Shi, Liyun
Li, Wenhua
Gui, Jian-Fang
Liu, Xing
Xiao, Wuhan
author_facet Deng, Hongyan
Jia, Shuke
Zhu, Chunchun
Hua, Jiale
Wang, Zixuan
Sun, Xueyi
Liu, Wen
Shi, Liyun
Li, Wenhua
Gui, Jian-Fang
Liu, Xing
Xiao, Wuhan
Deng, Hongyan
Jia, Shuke
Zhu, Chunchun
Hua, Jiale
Wang, Zixuan
Sun, Xueyi
Liu, Wen
Shi, Liyun
Li, Wenhua
Gui, Jian-Fang
Liu, Xing
Xiao, Wuhan
collection PubMed - marine biology
contents IRF3 attenuates hypoxia signaling by retaining HIF-α in the cytoplasm. Deng, Hongyan Jia, Shuke Zhu, Chunchun Hua, Jiale Wang, Zixuan Sun, Xueyi Liu, Wen Shi, Liyun Li, Wenhua Gui, Jian-Fang Liu, Xing Xiao, Wuhan Animals Hypoxia-Inducible Factor 1, alpha Subunit Signal Transduction Cytoplasm Interferon Regulatory Factor-3 Basic Helix-Loop-Helix Proteins Endothelial PAS Domain-Containing Protein 1 Mice Zebrafish Cell Hypoxia Humans HEK293 Cells Interferon regulatory factor 3 (IRF3) functions as a key transcription factor in the innate antiviral immune response, which depends on its nuclear localization. However, its function in the cytoplasm during non-infection states remains largely unknown. In this study, we found that resting cytoplasmic IRF3 interacts with hypoxia-inducible factor (HIF)-1α and HIF-2α, two master regulators of hypoxia signaling. This interaction retains HIF-α in the cytoplasm under hypoxic conditions, preventing it from exerting its transcription factor function and attenuating hypoxia signaling. Disruption of IRF3 in both mice and zebrafish resulted in increased expression of hypoxia response genes and enhanced tolerance to hypoxia. These findings suggest that, in the absence of viral infection, cytoplasmic IRF3 modulates hypoxia signaling by retaining HIF-α in the cytoplasm under hypoxic conditions.
format Artículo científico
id pubmed_41520336
institution PubMed
language en
publishDate 2026
publisher Cell reports
record_format pubmed
spellingShingle IRF3 attenuates hypoxia signaling by retaining HIF-α in the cytoplasm.
Deng, Hongyan
Jia, Shuke
Zhu, Chunchun
Hua, Jiale
Wang, Zixuan
Sun, Xueyi
Liu, Wen
Shi, Liyun
Li, Wenhua
Gui, Jian-Fang
Liu, Xing
Xiao, Wuhan
Animals
Hypoxia-Inducible Factor 1, alpha Subunit
Signal Transduction
Cytoplasm
Interferon Regulatory Factor-3
Basic Helix-Loop-Helix Proteins
Endothelial PAS Domain-Containing Protein 1
Mice
Zebrafish
Cell Hypoxia
Humans
HEK293 Cells
IRF3 attenuates hypoxia signaling by retaining HIF-α in the cytoplasm. Deng, Hongyan Jia, Shuke Zhu, Chunchun Hua, Jiale Wang, Zixuan Sun, Xueyi Liu, Wen Shi, Liyun Li, Wenhua Gui, Jian-Fang Liu, Xing Xiao, Wuhan Animals Hypoxia-Inducible Factor 1, alpha Subunit Signal Transduction Cytoplasm Interferon Regulatory Factor-3 Basic Helix-Loop-Helix Proteins Endothelial PAS Domain-Containing Protein 1 Mice Zebrafish Cell Hypoxia Humans HEK293 Cells Interferon regulatory factor 3 (IRF3) functions as a key transcription factor in the innate antiviral immune response, which depends on its nuclear localization. However, its function in the cytoplasm during non-infection states remains largely unknown. In this study, we found that resting cytoplasmic IRF3 interacts with hypoxia-inducible factor (HIF)-1α and HIF-2α, two master regulators of hypoxia signaling. This interaction retains HIF-α in the cytoplasm under hypoxic conditions, preventing it from exerting its transcription factor function and attenuating hypoxia signaling. Disruption of IRF3 in both mice and zebrafish resulted in increased expression of hypoxia response genes and enhanced tolerance to hypoxia. These findings suggest that, in the absence of viral infection, cytoplasmic IRF3 modulates hypoxia signaling by retaining HIF-α in the cytoplasm under hypoxic conditions.
title IRF3 attenuates hypoxia signaling by retaining HIF-α in the cytoplasm.
topic Animals
Hypoxia-Inducible Factor 1, alpha Subunit
Signal Transduction
Cytoplasm
Interferon Regulatory Factor-3
Basic Helix-Loop-Helix Proteins
Endothelial PAS Domain-Containing Protein 1
Mice
Zebrafish
Cell Hypoxia
Humans
HEK293 Cells
url https://pubmed.ncbi.nlm.nih.gov/41520336/