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Main Authors: Häring, Matthias, Zhang, Yuanshu, Zhang, Na, Allgeyer, Edward S, Richens, Jennifer H, Sirinakis, George, Lv, Zhiyi, St Johnston, Daniel, Wolf, Fred, Großhans, Jörg, Kong, Deqing
Format: Artículo científico
Language:en
Published: Biophysical journal 2026
Subjects:
Online Access:https://pubmed.ncbi.nlm.nih.gov/41548041/
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author Häring, Matthias
Zhang, Yuanshu
Zhang, Na
Allgeyer, Edward S
Richens, Jennifer H
Sirinakis, George
Lv, Zhiyi
St Johnston, Daniel
Wolf, Fred
Großhans, Jörg
Kong, Deqing
author_facet Häring, Matthias
Zhang, Yuanshu
Zhang, Na
Allgeyer, Edward S
Richens, Jennifer H
Sirinakis, George
Lv, Zhiyi
St Johnston, Daniel
Wolf, Fred
Großhans, Jörg
Kong, Deqing
Häring, Matthias
Zhang, Yuanshu
Zhang, Na
Allgeyer, Edward S
Richens, Jennifer H
Sirinakis, George
Lv, Zhiyi
St Johnston, Daniel
Wolf, Fred
Großhans, Jörg
Kong, Deqing
collection PubMed - marine biology
contents DNA-PAINT resolves E-cadherin-independent cross-junctional F-actin organization in Drosophila embryonic tissue. Häring, Matthias Zhang, Yuanshu Zhang, Na Allgeyer, Edward S Richens, Jennifer H Sirinakis, George Lv, Zhiyi St Johnston, Daniel Wolf, Fred Großhans, Jörg Kong, Deqing Animals Cadherins Actins Adherens Junctions Drosophila melanogaster Embryo, Nonmammalian DNA Cell junction remodeling is central to epithelial morphogenesis and tissue rheology and depends on the interplay between adhesion molecules and the actomyosin cortex. E-cadherin constitutes the molecular basis for epithelial cell adhesion, whereas cortical actomyosin plays a major role in intracellular force generation. However, the precise nanoscale organization and relationship between F-actin and E-cadherin at the cell interface still remain insufficiently understood. Here, we applied super-resolution DNA/peptide-PAINT microscopy to reveal the nanoscopic clustering of E-cadherin and its junctional distribution in relation to cortical F-actin at adherens junctions in the Drosophila embryonic epidermis. We were able to resolve distinct pairs of E-cadherin clusters approximately 45 nm apart on opposite sides of the adherens junctions. Intriguingly, these paired clusters were interspersed with unpaired clusters, lacking corresponding counterparts across the junction. We observed that cluster size, spatial arrangement, and cross-junction matching change during development and depend on N-glycosylation, a posttranslational modification affecting E-cadherin. Moreover, the organization of F-actin cortices between neighboring cells was found to be strongly correlated at junctions. Contrary to expectations, this intercellular F-actin correlation was observed independently of E-cadherin. Our study provides new insights into the nanoscale organization of adherens junctions, opening a window into the molecular mechanism of adhesion and mechanics of epithelial cells during morphogenesis.
format Artículo científico
id pubmed_41548041
institution PubMed
language en
publishDate 2026
publisher Biophysical journal
record_format pubmed
spellingShingle DNA-PAINT resolves E-cadherin-independent cross-junctional F-actin organization in Drosophila embryonic tissue.
Häring, Matthias
Zhang, Yuanshu
Zhang, Na
Allgeyer, Edward S
Richens, Jennifer H
Sirinakis, George
Lv, Zhiyi
St Johnston, Daniel
Wolf, Fred
Großhans, Jörg
Kong, Deqing
Animals
Cadherins
Actins
Adherens Junctions
Drosophila melanogaster
Embryo, Nonmammalian
DNA
DNA-PAINT resolves E-cadherin-independent cross-junctional F-actin organization in Drosophila embryonic tissue. Häring, Matthias Zhang, Yuanshu Zhang, Na Allgeyer, Edward S Richens, Jennifer H Sirinakis, George Lv, Zhiyi St Johnston, Daniel Wolf, Fred Großhans, Jörg Kong, Deqing Animals Cadherins Actins Adherens Junctions Drosophila melanogaster Embryo, Nonmammalian DNA Cell junction remodeling is central to epithelial morphogenesis and tissue rheology and depends on the interplay between adhesion molecules and the actomyosin cortex. E-cadherin constitutes the molecular basis for epithelial cell adhesion, whereas cortical actomyosin plays a major role in intracellular force generation. However, the precise nanoscale organization and relationship between F-actin and E-cadherin at the cell interface still remain insufficiently understood. Here, we applied super-resolution DNA/peptide-PAINT microscopy to reveal the nanoscopic clustering of E-cadherin and its junctional distribution in relation to cortical F-actin at adherens junctions in the Drosophila embryonic epidermis. We were able to resolve distinct pairs of E-cadherin clusters approximately 45 nm apart on opposite sides of the adherens junctions. Intriguingly, these paired clusters were interspersed with unpaired clusters, lacking corresponding counterparts across the junction. We observed that cluster size, spatial arrangement, and cross-junction matching change during development and depend on N-glycosylation, a posttranslational modification affecting E-cadherin. Moreover, the organization of F-actin cortices between neighboring cells was found to be strongly correlated at junctions. Contrary to expectations, this intercellular F-actin correlation was observed independently of E-cadherin. Our study provides new insights into the nanoscale organization of adherens junctions, opening a window into the molecular mechanism of adhesion and mechanics of epithelial cells during morphogenesis.
title DNA-PAINT resolves E-cadherin-independent cross-junctional F-actin organization in Drosophila embryonic tissue.
topic Animals
Cadherins
Actins
Adherens Junctions
Drosophila melanogaster
Embryo, Nonmammalian
DNA
url https://pubmed.ncbi.nlm.nih.gov/41548041/