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Main Authors: Khamineh, Yasaman, Panahi-Alanagh, Sanaz, Zolghadri, Samaneh, Mavaddatiyan, Laleh, Ryszkiel, Ireneusz, Stanek, Agata, Talkhabi, Mahmood
Format: Artículo científico
Language:en
Published: Cells 2026
Subjects:
Online Access:https://pubmed.ncbi.nlm.nih.gov/41677644/
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author Khamineh, Yasaman
Panahi-Alanagh, Sanaz
Zolghadri, Samaneh
Mavaddatiyan, Laleh
Ryszkiel, Ireneusz
Stanek, Agata
Talkhabi, Mahmood
author_facet Khamineh, Yasaman
Panahi-Alanagh, Sanaz
Zolghadri, Samaneh
Mavaddatiyan, Laleh
Ryszkiel, Ireneusz
Stanek, Agata
Talkhabi, Mahmood
Khamineh, Yasaman
Panahi-Alanagh, Sanaz
Zolghadri, Samaneh
Mavaddatiyan, Laleh
Ryszkiel, Ireneusz
Stanek, Agata
Talkhabi, Mahmood
collection PubMed - marine biology
contents Effects of N-Acetylcysteine and Alpha-Ketoglutarate on OVCAR3 Ovarian Cancer Cells: Insights from Integrative Bioinformatics and Experimental Validation. Khamineh, Yasaman Panahi-Alanagh, Sanaz Zolghadri, Samaneh Mavaddatiyan, Laleh Ryszkiel, Ireneusz Stanek, Agata Talkhabi, Mahmood Humans Female Ovarian Neoplasms Acetylcysteine Ketoglutaric Acids Cell Line, Tumor Cell Movement Apoptosis Computational Biology Cell Survival Gene Expression Regulation, Neoplastic Protein Interaction Maps Cell Proliferation Ovarian cancer remains one of the leading causes of cancer-related mortality among women, underscoring the need for novel combination strategies that effectively inhibit tumor cell growth while limiting adverse effects. N-acetylcysteine (NAC) and alpha-ketoglutarate (AKG) are biologically active compounds with reported anticancer properties; however, their combined effects in ovarian cancer are not well characterized. In this study, we applied an integrative approach combining network pharmacology analysis with in vitro experiments to investigate the effects of NAC and AKG on OVCAR3 ovarian cancer cells. Common molecular targets of NAC and AKG were identified by intersecting predicted compound targets with ovarian cancer-associated genes, followed by protein-protein interaction network construction and Gene Ontology and Kyoto Encyclopedia of Genes and Genomes pathway enrichment analyses. Experimental validation assessed the effects of NAC and AKG, alone and in combination, on cell viability, apoptosis, migration, and clonogenic capacity. Network analysis identified 70 shared target genes enriched in pathways related to apoptosis, cellular stress responses, and cell migration. In vitro experiments demonstrated that combined treatment with NAC (10 mM) and AKG (100 µM) significantly reduced cell viability, increased apoptotic cell death, and markedly suppressed cell migration and colony formation compared with single-agent treatments. Overall, these findings indicate that the combination of NAC and AKG exerts enhanced inhibitory effects on ovarian cancer cell growth and motility in vitro.
format Artículo científico
id pubmed_41677644
institution PubMed
language en
publishDate 2026
publisher Cells
record_format pubmed
spellingShingle Effects of N-Acetylcysteine and Alpha-Ketoglutarate on OVCAR3 Ovarian Cancer Cells: Insights from Integrative Bioinformatics and Experimental Validation.
Khamineh, Yasaman
Panahi-Alanagh, Sanaz
Zolghadri, Samaneh
Mavaddatiyan, Laleh
Ryszkiel, Ireneusz
Stanek, Agata
Talkhabi, Mahmood
Humans
Female
Ovarian Neoplasms
Acetylcysteine
Ketoglutaric Acids
Cell Line, Tumor
Cell Movement
Apoptosis
Computational Biology
Cell Survival
Gene Expression Regulation, Neoplastic
Protein Interaction Maps
Cell Proliferation
Effects of N-Acetylcysteine and Alpha-Ketoglutarate on OVCAR3 Ovarian Cancer Cells: Insights from Integrative Bioinformatics and Experimental Validation. Khamineh, Yasaman Panahi-Alanagh, Sanaz Zolghadri, Samaneh Mavaddatiyan, Laleh Ryszkiel, Ireneusz Stanek, Agata Talkhabi, Mahmood Humans Female Ovarian Neoplasms Acetylcysteine Ketoglutaric Acids Cell Line, Tumor Cell Movement Apoptosis Computational Biology Cell Survival Gene Expression Regulation, Neoplastic Protein Interaction Maps Cell Proliferation Ovarian cancer remains one of the leading causes of cancer-related mortality among women, underscoring the need for novel combination strategies that effectively inhibit tumor cell growth while limiting adverse effects. N-acetylcysteine (NAC) and alpha-ketoglutarate (AKG) are biologically active compounds with reported anticancer properties; however, their combined effects in ovarian cancer are not well characterized. In this study, we applied an integrative approach combining network pharmacology analysis with in vitro experiments to investigate the effects of NAC and AKG on OVCAR3 ovarian cancer cells. Common molecular targets of NAC and AKG were identified by intersecting predicted compound targets with ovarian cancer-associated genes, followed by protein-protein interaction network construction and Gene Ontology and Kyoto Encyclopedia of Genes and Genomes pathway enrichment analyses. Experimental validation assessed the effects of NAC and AKG, alone and in combination, on cell viability, apoptosis, migration, and clonogenic capacity. Network analysis identified 70 shared target genes enriched in pathways related to apoptosis, cellular stress responses, and cell migration. In vitro experiments demonstrated that combined treatment with NAC (10 mM) and AKG (100 µM) significantly reduced cell viability, increased apoptotic cell death, and markedly suppressed cell migration and colony formation compared with single-agent treatments. Overall, these findings indicate that the combination of NAC and AKG exerts enhanced inhibitory effects on ovarian cancer cell growth and motility in vitro.
title Effects of N-Acetylcysteine and Alpha-Ketoglutarate on OVCAR3 Ovarian Cancer Cells: Insights from Integrative Bioinformatics and Experimental Validation.
topic Humans
Female
Ovarian Neoplasms
Acetylcysteine
Ketoglutaric Acids
Cell Line, Tumor
Cell Movement
Apoptosis
Computational Biology
Cell Survival
Gene Expression Regulation, Neoplastic
Protein Interaction Maps
Cell Proliferation
url https://pubmed.ncbi.nlm.nih.gov/41677644/