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Main Authors: Du, Yueying, Deng, Yuhang, Liu, Ruixue, Yuan, Jialing, Wang, Yunxia, Wang, Su, Nie, Pin, Song, Xiaojun
Format: Artículo científico
Language:en
Published: Fish & shellfish immunology 2026
Subjects:
Online Access:https://pubmed.ncbi.nlm.nih.gov/41887303/
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author Du, Yueying
Deng, Yuhang
Liu, Ruixue
Yuan, Jialing
Wang, Yunxia
Wang, Su
Nie, Pin
Song, Xiaojun
author_facet Du, Yueying
Deng, Yuhang
Liu, Ruixue
Yuan, Jialing
Wang, Yunxia
Wang, Su
Nie, Pin
Song, Xiaojun
Du, Yueying
Deng, Yuhang
Liu, Ruixue
Yuan, Jialing
Wang, Yunxia
Wang, Su
Nie, Pin
Song, Xiaojun
collection PubMed - marine biology
contents Genome-wide identification, expression analysis and antiviral function of RIG-I-like receptors (RLRs) in basal chordate Branchiostoma japonicum. Du, Yueying Deng, Yuhang Liu, Ruixue Yuan, Jialing Wang, Yunxia Wang, Su Nie, Pin Song, Xiaojun Animals Immunity, Innate Lancelets Poly I-C Phylogeny DEAD Box Protein 58 Lipopolysaccharides Gene Expression Profiling Gene Expression Regulation The retinoic acid-inducible gene I (RIG-I)-like receptors (RLRs) serve as critical cytoplasmic RNA sensors and play key roles in antiviral innate immunity. However, the distribution, evolutionary dynamics, and antiviral functions of RLRs in basal chordates remain poorly understood. In this study, we investigated the distribution and evolutionary patterns of RLRs across species ranging from single-celled eukaryotes to mammals, and further examined their antiviral innate immune responses in the basal chordate Branchiostoma japonicum. Our results demonstrate that RLRs are widely present in metazoans from sponges to mammals: two subfamilies (RIG-I and Melanoma Differentiation-Associated protein 5, MDA5) in invertebrate and three subfamilies (RIG-I, MDA5 and Laboratory of Genetics and Physiology 2, LGP2) in vertebrate, with evolutionary features including gene duplication and loss, domain rearrangement, and positive selection in the evolutionary history. Acute immune challenge experiments revealed that expression of RLRs in B. japonicum was significantly altered following infection with poly(I:C), LPS and poly(dA:dT). Subcellular localization analysis indicated that both BjRIG-Ia and BjMDA5a are broadly distributed in the cytoplasm. Overexpression experiments showed that overexpression of BjMDA5a can upregulate the expression of several RLR signaling pathway genes, such as DHX58, TBK1, IFNα, IRF3, IRF7, ISG15, ATG12, and TNFα. In contrast, overexpression of BjRIG-Ia significantly downregulated the expression of DHX58, ATG12, IκBα, IRF3, IRF7, ISG15, p65, and p50. Given the established role of MDA5 in antiviral immunity, crystal violet staining assays demonstrated that BjMDA5a inhibited the proliferation of spring viremia of carp virus (SVCV) at the cellular level in a dose-dependent manner. This study provides valuable insights into the origin and evolution of antiviral regulatory networks in chordates.
format Artículo científico
id pubmed_41887303
institution PubMed
language en
publishDate 2026
publisher Fish & shellfish immunology
record_format pubmed
spellingShingle Genome-wide identification, expression analysis and antiviral function of RIG-I-like receptors (RLRs) in basal chordate Branchiostoma japonicum.
Du, Yueying
Deng, Yuhang
Liu, Ruixue
Yuan, Jialing
Wang, Yunxia
Wang, Su
Nie, Pin
Song, Xiaojun
Animals
Immunity, Innate
Lancelets
Poly I-C
Phylogeny
DEAD Box Protein 58
Lipopolysaccharides
Gene Expression Profiling
Gene Expression Regulation
Genome-wide identification, expression analysis and antiviral function of RIG-I-like receptors (RLRs) in basal chordate Branchiostoma japonicum. Du, Yueying Deng, Yuhang Liu, Ruixue Yuan, Jialing Wang, Yunxia Wang, Su Nie, Pin Song, Xiaojun Animals Immunity, Innate Lancelets Poly I-C Phylogeny DEAD Box Protein 58 Lipopolysaccharides Gene Expression Profiling Gene Expression Regulation The retinoic acid-inducible gene I (RIG-I)-like receptors (RLRs) serve as critical cytoplasmic RNA sensors and play key roles in antiviral innate immunity. However, the distribution, evolutionary dynamics, and antiviral functions of RLRs in basal chordates remain poorly understood. In this study, we investigated the distribution and evolutionary patterns of RLRs across species ranging from single-celled eukaryotes to mammals, and further examined their antiviral innate immune responses in the basal chordate Branchiostoma japonicum. Our results demonstrate that RLRs are widely present in metazoans from sponges to mammals: two subfamilies (RIG-I and Melanoma Differentiation-Associated protein 5, MDA5) in invertebrate and three subfamilies (RIG-I, MDA5 and Laboratory of Genetics and Physiology 2, LGP2) in vertebrate, with evolutionary features including gene duplication and loss, domain rearrangement, and positive selection in the evolutionary history. Acute immune challenge experiments revealed that expression of RLRs in B. japonicum was significantly altered following infection with poly(I:C), LPS and poly(dA:dT). Subcellular localization analysis indicated that both BjRIG-Ia and BjMDA5a are broadly distributed in the cytoplasm. Overexpression experiments showed that overexpression of BjMDA5a can upregulate the expression of several RLR signaling pathway genes, such as DHX58, TBK1, IFNα, IRF3, IRF7, ISG15, ATG12, and TNFα. In contrast, overexpression of BjRIG-Ia significantly downregulated the expression of DHX58, ATG12, IκBα, IRF3, IRF7, ISG15, p65, and p50. Given the established role of MDA5 in antiviral immunity, crystal violet staining assays demonstrated that BjMDA5a inhibited the proliferation of spring viremia of carp virus (SVCV) at the cellular level in a dose-dependent manner. This study provides valuable insights into the origin and evolution of antiviral regulatory networks in chordates.
title Genome-wide identification, expression analysis and antiviral function of RIG-I-like receptors (RLRs) in basal chordate Branchiostoma japonicum.
topic Animals
Immunity, Innate
Lancelets
Poly I-C
Phylogeny
DEAD Box Protein 58
Lipopolysaccharides
Gene Expression Profiling
Gene Expression Regulation
url https://pubmed.ncbi.nlm.nih.gov/41887303/