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Main Authors: Singh, Simran, Damodaran, Anusree, Goswami, Sanskriti, Lata, Manjul, Pasupuleti, Mukesh, Verma, Sonia
Format: Artículo científico
Language:en
Published: Scientific reports 2026
Subjects:
Online Access:https://pubmed.ncbi.nlm.nih.gov/41933141/
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author Singh, Simran
Damodaran, Anusree
Goswami, Sanskriti
Lata, Manjul
Pasupuleti, Mukesh
Verma, Sonia
author_facet Singh, Simran
Damodaran, Anusree
Goswami, Sanskriti
Lata, Manjul
Pasupuleti, Mukesh
Verma, Sonia
Singh, Simran
Damodaran, Anusree
Goswami, Sanskriti
Lata, Manjul
Pasupuleti, Mukesh
Verma, Sonia
collection PubMed - marine biology
contents Marine bacterium Stutzerimonas stutzeri mitigates Parkinson's disease pathology in C. elegans via ferroptosis modulation. Singh, Simran Damodaran, Anusree Goswami, Sanskriti Lata, Manjul Pasupuleti, Mukesh Verma, Sonia Animals Caenorhabditis elegans Parkinson Disease Ferroptosis Dopaminergic Neurons Disease Models, Animal alpha-Synuclein Neuroprotective Agents Lipid Peroxidation Parkinson's disease (PD) is a rapidly escalating neurodegenerative disorder marked by dopaminergic neurodegeneration, α-synuclein aggregation, and motor and non-motor impairments. Current therapies largely provide symptomatic relief and fail to prevent disease progression, underscoring the need for novel disease-modifying strategies. The marine biome has emerged as an unexplored reservoir of bioactive metabolites with neuroprotective potential, yet their therapeutic relevance in PD remains incompletely explored. Here, we report that Stutzerimonas stutzeri, a marine bacterium isolated from the Gulf of Mannar, exerts robust neuroprotective effects in Caenorhabditis elegans PD models. Dietary administration of S. stutzeri rescued dopaminergic neuronal loss, mitigated α-synuclein expression, and improved motor and sensory phenotypes. Mechanistic analyses revealed suppression of ferroptosis, evidenced by restoration of iron homeostasis, attenuation of lipid peroxidation, and recovery of ftn-1 expression. Our findings establish S. stutzeri as a previously unrecognized marine-derived therapeutic prospect for PD intervention and highlight ferroptosis modulation as a tractable therapeutic axis in neurodegeneration.
format Artículo científico
id pubmed_41933141
institution PubMed
language en
publishDate 2026
publisher Scientific reports
record_format pubmed
spellingShingle Marine bacterium Stutzerimonas stutzeri mitigates Parkinson's disease pathology in C. elegans via ferroptosis modulation.
Singh, Simran
Damodaran, Anusree
Goswami, Sanskriti
Lata, Manjul
Pasupuleti, Mukesh
Verma, Sonia
Animals
Caenorhabditis elegans
Parkinson Disease
Ferroptosis
Dopaminergic Neurons
Disease Models, Animal
alpha-Synuclein
Neuroprotective Agents
Lipid Peroxidation
Marine bacterium Stutzerimonas stutzeri mitigates Parkinson's disease pathology in C. elegans via ferroptosis modulation. Singh, Simran Damodaran, Anusree Goswami, Sanskriti Lata, Manjul Pasupuleti, Mukesh Verma, Sonia Animals Caenorhabditis elegans Parkinson Disease Ferroptosis Dopaminergic Neurons Disease Models, Animal alpha-Synuclein Neuroprotective Agents Lipid Peroxidation Parkinson's disease (PD) is a rapidly escalating neurodegenerative disorder marked by dopaminergic neurodegeneration, α-synuclein aggregation, and motor and non-motor impairments. Current therapies largely provide symptomatic relief and fail to prevent disease progression, underscoring the need for novel disease-modifying strategies. The marine biome has emerged as an unexplored reservoir of bioactive metabolites with neuroprotective potential, yet their therapeutic relevance in PD remains incompletely explored. Here, we report that Stutzerimonas stutzeri, a marine bacterium isolated from the Gulf of Mannar, exerts robust neuroprotective effects in Caenorhabditis elegans PD models. Dietary administration of S. stutzeri rescued dopaminergic neuronal loss, mitigated α-synuclein expression, and improved motor and sensory phenotypes. Mechanistic analyses revealed suppression of ferroptosis, evidenced by restoration of iron homeostasis, attenuation of lipid peroxidation, and recovery of ftn-1 expression. Our findings establish S. stutzeri as a previously unrecognized marine-derived therapeutic prospect for PD intervention and highlight ferroptosis modulation as a tractable therapeutic axis in neurodegeneration.
title Marine bacterium Stutzerimonas stutzeri mitigates Parkinson's disease pathology in C. elegans via ferroptosis modulation.
topic Animals
Caenorhabditis elegans
Parkinson Disease
Ferroptosis
Dopaminergic Neurons
Disease Models, Animal
alpha-Synuclein
Neuroprotective Agents
Lipid Peroxidation
url https://pubmed.ncbi.nlm.nih.gov/41933141/