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Autores principales: Stuckless, Emily E, Gai, Lian S, Slattery, Samuel S, Dempsey, Kira H, Browne, Tyler S, Gloor, Gregory B, Edgell, David R
Formato: Artículo científico
Lenguaje:en
Publicado: ACS synthetic biology 2026
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Acceso en línea:https://pubmed.ncbi.nlm.nih.gov/41979903/
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author Stuckless, Emily E
Gai, Lian S
Slattery, Samuel S
Dempsey, Kira H
Browne, Tyler S
Gloor, Gregory B
Edgell, David R
author_facet Stuckless, Emily E
Gai, Lian S
Slattery, Samuel S
Dempsey, Kira H
Browne, Tyler S
Gloor, Gregory B
Edgell, David R
Stuckless, Emily E
Gai, Lian S
Slattery, Samuel S
Dempsey, Kira H
Browne, Tyler S
Gloor, Gregory B
Edgell, David R
collection PubMed - marine biology
contents PHYCUT: Scalable Multiplex CRISPR/Cas9 Editing for Genome Engineering in the Diatom . Stuckless, Emily E Gai, Lian S Slattery, Samuel S Dempsey, Kira H Browne, Tyler S Gloor, Gregory B Edgell, David R Diatoms CRISPR-Cas Systems Gene Editing Glycosylation Plasmids Fucose Diatoms are globally significant microalgae that contribute ∼20% of oxygen production and exhibit remarkable metabolic diversity. The marine diatom has emerged as a promising synthetic biology platform for the bioproduction of recombinant proteins, supported by a human-like -linked glycosylation pathway. However, its α(1,3)-linked core fucose is potentially immunogenic in humans and thus limits its biopharmaceutical applications. One hurdle to efficient genome engineering in is the lack of a robust system for simultaneous CRISPR/Cas9 editing at multiple sites. To overcome this limitation, we develop PHYCUT ( Cs4-as9 mltiplex ool), a versatile plasmid-based CRISPR/Cas9 system that uses the Csy4 endoribonuclease to process multiguide RNA arrays. To highlight PHYCUT applications, we demonstrate multiplex editing of all three genes responsible for α(1,3) fucosylation in , yielding strains with reduced fucosylation of secreted proteins. PHYCUT enables facile, multiplexed genome engineering in diatoms and provides a foundation for humanizing the glycosylation pathway to support next-generation algal biotechnology.
format Artículo científico
id pubmed_41979903
institution PubMed
language en
publishDate 2026
publisher ACS synthetic biology
record_format pubmed
spellingShingle PHYCUT: Scalable Multiplex CRISPR/Cas9 Editing for Genome Engineering in the Diatom .
Stuckless, Emily E
Gai, Lian S
Slattery, Samuel S
Dempsey, Kira H
Browne, Tyler S
Gloor, Gregory B
Edgell, David R
Diatoms
CRISPR-Cas Systems
Gene Editing
Glycosylation
Plasmids
Fucose
PHYCUT: Scalable Multiplex CRISPR/Cas9 Editing for Genome Engineering in the Diatom . Stuckless, Emily E Gai, Lian S Slattery, Samuel S Dempsey, Kira H Browne, Tyler S Gloor, Gregory B Edgell, David R Diatoms CRISPR-Cas Systems Gene Editing Glycosylation Plasmids Fucose Diatoms are globally significant microalgae that contribute ∼20% of oxygen production and exhibit remarkable metabolic diversity. The marine diatom has emerged as a promising synthetic biology platform for the bioproduction of recombinant proteins, supported by a human-like -linked glycosylation pathway. However, its α(1,3)-linked core fucose is potentially immunogenic in humans and thus limits its biopharmaceutical applications. One hurdle to efficient genome engineering in is the lack of a robust system for simultaneous CRISPR/Cas9 editing at multiple sites. To overcome this limitation, we develop PHYCUT ( Cs4-as9 mltiplex ool), a versatile plasmid-based CRISPR/Cas9 system that uses the Csy4 endoribonuclease to process multiguide RNA arrays. To highlight PHYCUT applications, we demonstrate multiplex editing of all three genes responsible for α(1,3) fucosylation in , yielding strains with reduced fucosylation of secreted proteins. PHYCUT enables facile, multiplexed genome engineering in diatoms and provides a foundation for humanizing the glycosylation pathway to support next-generation algal biotechnology.
title PHYCUT: Scalable Multiplex CRISPR/Cas9 Editing for Genome Engineering in the Diatom .
topic Diatoms
CRISPR-Cas Systems
Gene Editing
Glycosylation
Plasmids
Fucose
url https://pubmed.ncbi.nlm.nih.gov/41979903/