Aβ ‐induced excessive mitochondrial fission drives type H blood vessels injury to aggravate bone loss in APP/PS1 mice with Alzheimer's diseases

Documenti analoghi

• Overexpression of mitochondrial fission or mitochondrial fusion genes enhances resilience and extends longevity
  di: Annika Traa, et al.
  Pubblicazione: (2024)
• Single‐cell mitochondrial sequencing reveals low‐frequency mitochondrial mutations in naturally aging mice
  di: Fuyan Liu, et al.
  Pubblicazione: (2024)
• Piezo1 exacerbates inflammation‐induced cartilaginous endplate degeneration by activating mitochondrial fission via the Ca2+/CaMKII/Drp1 axis
  di: Zhidi Lin, et al.
  Pubblicazione: (2024)
• Alpha‐Ketoglutarate Ameliorates Synaptic Plasticity Deficits in APP / PS1 Mice Model of Alzheimer's Disease
  di: Sheeja Navakkode, et al.
  Pubblicazione: (2025)
• APOE4 Exacerbates Alzheimer‐Like Pathologies and Cognitive Deficits Induced by Blood‐Derived Aβ in a Mouse Model
  di: Zhong‐Yuan Yu, et al.
  Pubblicazione: (2025)