Ginsenoside compound K up‐regulates β‐subunits of long‐chain L‐3‐hydroxyacyl‐CoA (HADHB) to promote fatty acid oxidation recovery and improve the balance of macrophage polarization in mice with ulcerative colitis

Documenti analoghi

• Ginsenoside Compound K Inhibits Wnt/β‐Catenin Signaling and Anti‐Angiogenesis, Alleviating Joint Injury in Rats With Adjuvant Arthritis
  di: Xiujin Xu, et al.
  Pubblicazione: (2025)
• Thiamine deficiency aggravates experimental colitis in mice by promoting glycolytic reprogramming in macrophages
  di: Xiaohua Pan, et al.
  Pubblicazione: (2025)
• Loss of Bcl‐3 regulates macrophage polarization by promoting macrophage glycolysis
  di: Shengnan Liu, et al.
  Pubblicazione: (2024)
• Engineered probiotic alleviates ulcerative colitis by inhibiting M1 macrophage polarization via glycolytic reprogramming
  di: Chaoqun Lv, et al.
  Pubblicazione: (2025)
• Epimedium polysaccharides ameliorate ulcerative colitis by inhibiting oxidative stress and regulating autophagy
  di: Linxian Zhao, et al.
  Pubblicazione: (2024)