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Main Authors: Risha Annamraju, Madison S. Owens, Anita Thyagarajan, Danielle A. Corbin, Catherine M. T. Sherwin, Jade Bryant, Garrett W. Fisher, Winston R. Owens, Alycia Ketter, Aadil Umerani, Craig A. Rohan, Michael G. Kemp, Robyn K. Crow, Jeffrey B. Travers
Format: Artículo Open Access
Published: Wiley 2025
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Online Access:https://onlinelibrary.wiley.com/doi/10.1111/php.70058
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author Risha Annamraju
Madison S. Owens
Anita Thyagarajan
Danielle A. Corbin
Catherine M. T. Sherwin
Jade Bryant
Garrett W. Fisher
Winston R. Owens
Alycia Ketter
Aadil Umerani
Craig A. Rohan
Michael G. Kemp
Robyn K. Crow
Jeffrey B. Travers
author_facet Risha Annamraju
Madison S. Owens
Anita Thyagarajan
Danielle A. Corbin
Catherine M. T. Sherwin
Jade Bryant
Garrett W. Fisher
Winston R. Owens
Alycia Ketter
Aadil Umerani
Craig A. Rohan
Michael G. Kemp
Robyn K. Crow
Jeffrey B. Travers
Risha Annamraju
Madison S. Owens
Anita Thyagarajan
Danielle A. Corbin
Catherine M. T. Sherwin
Jade Bryant
Garrett W. Fisher
Winston R. Owens
Alycia Ketter
Aadil Umerani
Craig A. Rohan
Michael G. Kemp
Robyn K. Crow
Jeffrey B. Travers
collection Wiley Open Access
contents Possible involvement of keratinocyte‐derived microvesicle particles in human photosensitivity disorders Risha Annamraju Madison S. Owens Anita Thyagarajan Danielle A. Corbin Catherine M. T. Sherwin Jade Bryant Garrett W. Fisher Winston R. Owens Alycia Ketter Aadil Umerani Craig A. Rohan Michael G. Kemp Robyn K. Crow Jeffrey B. Travers Photochemistry and Photobiology Abstract Previous murine studies have implicated acid sphingomyelinase (aSMase)‐generated subcellular microvesicle particles (MVP) in photosensitivity. Objective: The current double‐blinded placebo‐controlled studies examined if a single localized ultraviolet B radiation (UVB) treatment generated more MVP in human subjects with self‐identified photosensitivity versus normal controls. A topical 4% formulation of the aSMase inhibitor imipramine applied immediately after UVB blocked the MVP release and erythema responses. Erythema responses at 24 and 72 h in response to multiple UVB fluences and minimal erythema doses (MED) at 24 h and effects of imipramine were also tested. Small cohorts of 10 adult self‐identified photosensitive subjects and 12 controls were enrolled in these pilot studies which revealed increased levels of skin MVP in UVB‐treated photosensitive subjects over controls which correlated with MED values. Moreover, post‐UVB application of imipramine blunted UVB‐induced MVP responses as well as tended to diminish erythema levels at 4 h but not at 24 or 72 h in photosensitive patients. Though limited by low numbers of self‐identified subjects, these pilot studies provide some support for the hypothesis that MVP could be involved in multiple types of human photosensitivity responses and suggest aSMase inhibition as a potential therapeutic strategy. 10.1111/php.70058 http://creativecommons.org/licenses/by-nc-nd/4.0/
doi_str_mv 10.1111/php.70058
format Artículo Open Access
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institution Wiley Open Access
license_str_mv http://creativecommons.org/licenses/by-nc-nd/4.0/
publishDate 2025
publisher Wiley
record_format wiley_oa
spellingShingle Possible involvement of keratinocyte‐derived microvesicle particles in human photosensitivity disorders
Risha Annamraju
Madison S. Owens
Anita Thyagarajan
Danielle A. Corbin
Catherine M. T. Sherwin
Jade Bryant
Garrett W. Fisher
Winston R. Owens
Alycia Ketter
Aadil Umerani
Craig A. Rohan
Michael G. Kemp
Robyn K. Crow
Jeffrey B. Travers
Photochemistry and Photobiology
Possible involvement of keratinocyte‐derived microvesicle particles in human photosensitivity disorders Risha Annamraju Madison S. Owens Anita Thyagarajan Danielle A. Corbin Catherine M. T. Sherwin Jade Bryant Garrett W. Fisher Winston R. Owens Alycia Ketter Aadil Umerani Craig A. Rohan Michael G. Kemp Robyn K. Crow Jeffrey B. Travers Photochemistry and Photobiology Abstract Previous murine studies have implicated acid sphingomyelinase (aSMase)‐generated subcellular microvesicle particles (MVP) in photosensitivity. Objective: The current double‐blinded placebo‐controlled studies examined if a single localized ultraviolet B radiation (UVB) treatment generated more MVP in human subjects with self‐identified photosensitivity versus normal controls. A topical 4% formulation of the aSMase inhibitor imipramine applied immediately after UVB blocked the MVP release and erythema responses. Erythema responses at 24 and 72 h in response to multiple UVB fluences and minimal erythema doses (MED) at 24 h and effects of imipramine were also tested. Small cohorts of 10 adult self‐identified photosensitive subjects and 12 controls were enrolled in these pilot studies which revealed increased levels of skin MVP in UVB‐treated photosensitive subjects over controls which correlated with MED values. Moreover, post‐UVB application of imipramine blunted UVB‐induced MVP responses as well as tended to diminish erythema levels at 4 h but not at 24 or 72 h in photosensitive patients. Though limited by low numbers of self‐identified subjects, these pilot studies provide some support for the hypothesis that MVP could be involved in multiple types of human photosensitivity responses and suggest aSMase inhibition as a potential therapeutic strategy. 10.1111/php.70058 http://creativecommons.org/licenses/by-nc-nd/4.0/
title Possible involvement of keratinocyte‐derived microvesicle particles in human photosensitivity disorders
topic Photochemistry and Photobiology
url https://onlinelibrary.wiley.com/doi/10.1111/php.70058