Rescue of secretion of rare‐disease‐associated misfolded mutant glycoproteins in UGGT1 knock‐out mammalian cells

Documenti analoghi

• Endoglin mutants retained in the endoplasmic reticulum exacerbate loss of function in hereditary hemorrhagic telangiectasia type 1 (HHT1) by exerting dominant negative effects on the wild type allele
  di: Nesrin Gariballa, et al.
  Pubblicazione: (2024)
• Implementation of Adaptive Traffic Lights to Reduce Traffic Congestion at Intersections through Efficient Strategies for Selective Use of Detectors
  di: Erwin Romero, R., et al.
  Pubblicazione: (2026)
• TRIP RATES FOR CONDOMINIUM CONSTRUCTION PROJECT
  di: Wirach Hirun
  Pubblicazione: (2015)
• Disease‐Associated Factors at the Endoplasmic Reticulum–Golgi Interface
  di: Miharu Maeda, et al.
  Pubblicazione: (2025)
• Strain‐Dependent Immune Signaling by Small Extracellular Vesicles Derived From Trypanosoma cruzi ‐Infected Macrophages
  di: Andrea C. Mesías, et al.
  Pubblicazione: (2025)