Saved in:
Bibliographic Details
Main Authors: Hernandez-Muñoz, Inmaculada, Elisabeth Figuerola, Sánchez-Molina, Sara, Rodríguez, Eva, Ana Isabel Fernández-Mariño, Pardo-Pastor, Carlos, Bahamonde Santos, Maria Isabel, José M Fernández-Fernández, Daniel J García-Domínguez, Hontecillas-Prieto, Lourdes, Lavarino, Cinzia, Carcaboso, Angel, Carmen de Torres, Tirado, Oscar M, de Alava, Enrique, Mora, Jaume
Format: Recurso digital
Language:
Published: Zenodo 2016
Online Access:https://doi.org/10.18632/oncotarget.10092
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1866901859672784896
author Hernandez-Muñoz, Inmaculada
Elisabeth Figuerola
Sánchez-Molina, Sara
Rodríguez, Eva
Ana Isabel Fernández-Mariño
Pardo-Pastor, Carlos
Bahamonde Santos, Maria Isabel
José M Fernández-Fernández
Daniel J García-Domínguez
Hontecillas-Prieto, Lourdes
Lavarino, Cinzia
Carcaboso, Angel
Carmen de Torres
Tirado, Oscar M
de Alava, Enrique
Mora, Jaume
author_facet Hernandez-Muñoz, Inmaculada
Elisabeth Figuerola
Sánchez-Molina, Sara
Rodríguez, Eva
Ana Isabel Fernández-Mariño
Pardo-Pastor, Carlos
Bahamonde Santos, Maria Isabel
José M Fernández-Fernández
Daniel J García-Domínguez
Hontecillas-Prieto, Lourdes
Lavarino, Cinzia
Carcaboso, Angel
Carmen de Torres
Tirado, Oscar M
de Alava, Enrique
Mora, Jaume
contents
format Recurso digital
id zenodo_https___doi_org_10_18632_oncotarget_10092
institution Zenodo
language
publishDate 2016
publisher Zenodo
record_format zenodo
spellingShingle RING1B contributes to Ewing sarcoma development by repressing the NaV1.6 sodium channel and the NF-κB pathway, independently of the fusion oncoprotein
Hernandez-Muñoz, Inmaculada
Elisabeth Figuerola
Sánchez-Molina, Sara
Rodríguez, Eva
Ana Isabel Fernández-Mariño
Pardo-Pastor, Carlos
Bahamonde Santos, Maria Isabel
José M Fernández-Fernández
Daniel J García-Domínguez
Hontecillas-Prieto, Lourdes
Lavarino, Cinzia
Carcaboso, Angel
Carmen de Torres
Tirado, Oscar M
de Alava, Enrique
Mora, Jaume

title RING1B contributes to Ewing sarcoma development by repressing the NaV1.6 sodium channel and the NF-κB pathway, independently of the fusion oncoprotein
url https://doi.org/10.18632/oncotarget.10092