The Clinical Presentation of Thrombotic Microangiopathy

Fuente: Zenodo
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Autori principali: Guliko Kiliptari, Nino Agladze
Natura: Recurso digital
Lingua:Antico inglese
Pubblicazione: Zenodo 2025
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author Guliko Kiliptari
Nino Agladze
author_facet Guliko Kiliptari
Nino Agladze
contents <p>Thrombotic microangiopathy (TMA) is characterized by thrombocytopenia, microangiopathic haemolytic anaemia and end organ damage. Microangiopathic haemolytic anaemia is caused by red blood cell fragmentation in the microvasculature, with schistocytes seen on peripheral blood film. Lactate dehydrogenase (LDH) is raised due to tissue ischemia and cell lysis. Low plasma haptoglobin is a marker of haemolysis as it binds to free haemoglobin and the complex is cleared by macrophages. Coombs test is gener negative. Renal involvement is common to most TMAs due to its vulnerability to occlusion and endothelial damage.</p>
format Recurso digital
id zenodo_https___doi_org_10_5281_zenodo_15093714
institution Zenodo
language ang
publishDate 2025
publisher Zenodo
record_format zenodo
spellingShingle The Clinical Presentation of Thrombotic Microangiopathy
Guliko Kiliptari
Nino Agladze
HUS, renal replacement therapy, coma, vena cava thrombosis
<p>Thrombotic microangiopathy (TMA) is characterized by thrombocytopenia, microangiopathic haemolytic anaemia and end organ damage. Microangiopathic haemolytic anaemia is caused by red blood cell fragmentation in the microvasculature, with schistocytes seen on peripheral blood film. Lactate dehydrogenase (LDH) is raised due to tissue ischemia and cell lysis. Low plasma haptoglobin is a marker of haemolysis as it binds to free haemoglobin and the complex is cleared by macrophages. Coombs test is gener negative. Renal involvement is common to most TMAs due to its vulnerability to occlusion and endothelial damage.</p>
title The Clinical Presentation of Thrombotic Microangiopathy
topic HUS, renal replacement therapy, coma, vena cava thrombosis
url https://doi.org/10.5281/zenodo.15093714