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| Format: | Recurso digital |
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Zenodo
2025
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| Online Access: | https://doi.org/10.5281/zenodo.15527143 |
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| _version_ | 1866901119985254400 |
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| author | Mamoor, Shahan |
| author_facet | Mamoor, Shahan |
| contents | <p>We provide here transcriptome data associated with genetic modulation of the BRCA1-associated DNA damage pathway that demonstrates a role for ATM in Xist activation and repression found in cancer and in cells and tissues resulting from tumor suppressor mutation. The data support a general role for Xist control by HR and non-homologous end joining repair pathways mediated by H2AX, 53bp1, BRCA1, Rif1, and NBS complex component Mre11. </p> |
| format | Recurso digital |
| id | zenodo_https___doi_org_10_5281_zenodo_15527143 |
| institution | Zenodo |
| language | |
| publishDate | 2025 |
| publisher | Zenodo |
| record_format | zenodo |
| spellingShingle | ATM defects result in Xist modulation. Mamoor, Shahan <p>We provide here transcriptome data associated with genetic modulation of the BRCA1-associated DNA damage pathway that demonstrates a role for ATM in Xist activation and repression found in cancer and in cells and tissues resulting from tumor suppressor mutation. The data support a general role for Xist control by HR and non-homologous end joining repair pathways mediated by H2AX, 53bp1, BRCA1, Rif1, and NBS complex component Mre11. </p> |
| title | ATM defects result in Xist modulation. |
| url | https://doi.org/10.5281/zenodo.15527143 |