Saved in:
| Main Authors: | , , |
|---|---|
| Format: | Recurso digital |
| Language: | English |
| Published: |
Zenodo
2025
|
| Online Access: | https://doi.org/10.5281/zenodo.16662863 |
| Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
| _version_ | 1866902171757314048 |
|---|---|
| author | Diamandis, Carolina Balaskas, Alexandros Josef, Merab |
| author_facet | Diamandis, Carolina Balaskas, Alexandros Josef, Merab |
| contents | <p><strong>Postprandial ventricular extrasystoles represent a complex interplay of hemodynamic, metabolic,</strong></p> <p><strong>and autonomic mechanisms that create a predictable window of heightened arrhythmogenic</strong></p> <p><strong>vulnerability 30–60 minutes after meals. In patients with chronic insomnia and catecholamine</strong></p> <p><strong>dysregulation, these physiological responses become pathologically exaggerated and prolonged,</strong></p> <p><strong>significantly increasing the risk of clinically relevant arrhythmias. This paper explores the underlying</strong></p> <p><strong>mechanisms — including hemodynamic stress, macronutrient-specific metabolic pathways, gastro-</strong></p> <p><strong>cardiac reflexes, and glucose-catecholamine interactions — as well as the impact of sleep deprivation</strong></p> <p><strong>on sustained sympathetic dominance. Clinically, the distinction between benign VES in structurally</strong></p> <p><strong>normal hearts and pathological patterns requiring further evaluation is critical. Therapeutic strategies</strong></p> <p><strong>should adopt a multidimensional approach, combining sleep restoration, nutritional modifications, and</strong></p> <p><strong>autonomic function monitoring. Understanding the temporal patterns and pathophysiological drivers of</strong></p> <p><strong>postprandial arrhythmias provides actionable insights for managing complex patients presenting with</strong></p> <p><strong>postprandial cardiac symptoms.</strong></p> |
| format | Recurso digital |
| id | zenodo_https___doi_org_10_5281_zenodo_16662863 |
| institution | Zenodo |
| language | eng |
| publishDate | 2025 |
| publisher | Zenodo |
| record_format | zenodo |
| spellingShingle | Postprandial Ventricular Arrhythmias in patient suffering from H63D Syndrome Types 1, 2 and 3 (Oshtoran Syndrome) Diamandis, Carolina Balaskas, Alexandros Josef, Merab <p><strong>Postprandial ventricular extrasystoles represent a complex interplay of hemodynamic, metabolic,</strong></p> <p><strong>and autonomic mechanisms that create a predictable window of heightened arrhythmogenic</strong></p> <p><strong>vulnerability 30–60 minutes after meals. In patients with chronic insomnia and catecholamine</strong></p> <p><strong>dysregulation, these physiological responses become pathologically exaggerated and prolonged,</strong></p> <p><strong>significantly increasing the risk of clinically relevant arrhythmias. This paper explores the underlying</strong></p> <p><strong>mechanisms — including hemodynamic stress, macronutrient-specific metabolic pathways, gastro-</strong></p> <p><strong>cardiac reflexes, and glucose-catecholamine interactions — as well as the impact of sleep deprivation</strong></p> <p><strong>on sustained sympathetic dominance. Clinically, the distinction between benign VES in structurally</strong></p> <p><strong>normal hearts and pathological patterns requiring further evaluation is critical. Therapeutic strategies</strong></p> <p><strong>should adopt a multidimensional approach, combining sleep restoration, nutritional modifications, and</strong></p> <p><strong>autonomic function monitoring. Understanding the temporal patterns and pathophysiological drivers of</strong></p> <p><strong>postprandial arrhythmias provides actionable insights for managing complex patients presenting with</strong></p> <p><strong>postprandial cardiac symptoms.</strong></p> |
| title | Postprandial Ventricular Arrhythmias in patient suffering from H63D Syndrome Types 1, 2 and 3 (Oshtoran Syndrome) |
| url | https://doi.org/10.5281/zenodo.16662863 |