GUILLAIN–BARRÉ SYNDROME: COMPREHENSIVE REVIEW OF PATHOGENESIS, CLINICAL SPECTRUM, DIAGNOSIS, AND MANAGEMENT
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| Formato: | Recurso digital |
| Lenguaje: | Idioma anglosajón |
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Zenodo
2025
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| _version_ | 1866901867688099840 |
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| author | RAJENDIRAN, KAMESH |
| author_facet | RAJENDIRAN, KAMESH |
| contents | <p>Guillain–Barré Syndrome (GBS) is an acute immune-mediated polyradiculoneuropathy characterized by rapidly progressive, symmetrical weakness of the limbs with areflexia. It is the most common cause of acute flaccid paralysis worldwide after the eradication of poliomyelitis. The disease involves an aberrant immune response leading to demyelination or axonal damage of peripheral nerves, often following infections like Campylobacter jejuni, cytomegalovirus, Epstein-Barr virus, or influenza virus. Diagnosis relies on clinical features, cerebrospinal fluid (CSF) analysis showing albuminocytologic dissociation, and nerve conduction studies. Management focuses on immunotherapy (IVIg or plasma exchange) and supportive care. Early diagnosis and treatment significantly improve outcomes.</p> <p>Guillain–Barré Syndrome (GBS) is an acute immune-mediated polyradiculoneuropathy characterized by rapidly progressive, symmetrical weakness of the limbs with areflexia. It is the most common cause of acute flaccid paralysis worldwide after the eradication of poliomyelitis. The disease involves an aberrant immune response leading to demyelination or axonal damage of peripheral nerves, often following infections like Campylobacter jejuni, cytomegalovirus, Epstein-Barr virus, or influenza virus. Diagnosis relies on clinical features, cerebrospinal fluid (CSF) analysis showing albuminocytologic dissociation, and nerve conduction studies. Management focuses on immunotherapy (IVIg or plasma exchange) and supportive care. Early diagnosis and treatment significantly improve outcomes.</p> <p> </p> |
| format | Recurso digital |
| id | zenodo_https___doi_org_10_5281_zenodo_17496657 |
| institution | Zenodo |
| language | ang |
| publishDate | 2025 |
| publisher | Zenodo |
| record_format | zenodo |
| spellingShingle | GUILLAIN–BARRÉ SYNDROME: COMPREHENSIVE REVIEW OF PATHOGENESIS, CLINICAL SPECTRUM, DIAGNOSIS, AND MANAGEMENT RAJENDIRAN, KAMESH Neurosurgery <p>Guillain–Barré Syndrome (GBS) is an acute immune-mediated polyradiculoneuropathy characterized by rapidly progressive, symmetrical weakness of the limbs with areflexia. It is the most common cause of acute flaccid paralysis worldwide after the eradication of poliomyelitis. The disease involves an aberrant immune response leading to demyelination or axonal damage of peripheral nerves, often following infections like Campylobacter jejuni, cytomegalovirus, Epstein-Barr virus, or influenza virus. Diagnosis relies on clinical features, cerebrospinal fluid (CSF) analysis showing albuminocytologic dissociation, and nerve conduction studies. Management focuses on immunotherapy (IVIg or plasma exchange) and supportive care. Early diagnosis and treatment significantly improve outcomes.</p> <p>Guillain–Barré Syndrome (GBS) is an acute immune-mediated polyradiculoneuropathy characterized by rapidly progressive, symmetrical weakness of the limbs with areflexia. It is the most common cause of acute flaccid paralysis worldwide after the eradication of poliomyelitis. The disease involves an aberrant immune response leading to demyelination or axonal damage of peripheral nerves, often following infections like Campylobacter jejuni, cytomegalovirus, Epstein-Barr virus, or influenza virus. Diagnosis relies on clinical features, cerebrospinal fluid (CSF) analysis showing albuminocytologic dissociation, and nerve conduction studies. Management focuses on immunotherapy (IVIg or plasma exchange) and supportive care. Early diagnosis and treatment significantly improve outcomes.</p> <p> </p> |
| title | GUILLAIN–BARRÉ SYNDROME: COMPREHENSIVE REVIEW OF PATHOGENESIS, CLINICAL SPECTRUM, DIAGNOSIS, AND MANAGEMENT |
| topic | Neurosurgery |
| url | https://doi.org/10.5281/zenodo.17496657 |