Inflammation-Driven Postsynaptic Glutamatergic Dysfunction in Treatment-Resistant Depression: An Integrated Hypothesis Supported by Meta-Analytic Evidence and Novel Clinical Illustrations

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Main Author: Abdalmalik, Dafaallah
Format: Recurso digital
Published: Zenodo 2025
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author Abdalmalik, Dafaallah
author_facet Abdalmalik, Dafaallah
contents <p>A groundbreaking integrated hypothesis on Treatment-Resistant Depression (TRD), reviewed and noted as an 'interesting piece' by Professor Mario Maj. This research explores the Inflammation-Glutamate Axis, providing a mechanistic explanation for why conventional antidepressants fail. By bridging meta-analytic data with remarkable clinical transformations—where BDI scores dropped from 35 to 3—this work offers a novel roadmap for neuropsychiatric recovery. This upload includes the full manuscript and supplementary clinical data</p>
format Recurso digital
id zenodo_https___doi_org_10_5281_zenodo_20040535
institution Zenodo
language
publishDate 2025
publisher Zenodo
record_format zenodo
spellingShingle Inflammation-Driven Postsynaptic Glutamatergic Dysfunction in Treatment-Resistant Depression: An Integrated Hypothesis Supported by Meta-Analytic Evidence and Novel Clinical Illustrations
Abdalmalik, Dafaallah
Neuropsychiatry
Depressive Disorder
Astrocyte dysfunction
* Treatment-Resistant Depression
Glutamate hypothesis
Limitations of the Monoamine Hypothesis
Glutamate Excitotoxicity
Downregulation of Postsynaptic Receptors
<p>A groundbreaking integrated hypothesis on Treatment-Resistant Depression (TRD), reviewed and noted as an 'interesting piece' by Professor Mario Maj. This research explores the Inflammation-Glutamate Axis, providing a mechanistic explanation for why conventional antidepressants fail. By bridging meta-analytic data with remarkable clinical transformations—where BDI scores dropped from 35 to 3—this work offers a novel roadmap for neuropsychiatric recovery. This upload includes the full manuscript and supplementary clinical data</p>
title Inflammation-Driven Postsynaptic Glutamatergic Dysfunction in Treatment-Resistant Depression: An Integrated Hypothesis Supported by Meta-Analytic Evidence and Novel Clinical Illustrations
topic Neuropsychiatry
Depressive Disorder
Astrocyte dysfunction
* Treatment-Resistant Depression
Glutamate hypothesis
Limitations of the Monoamine Hypothesis
Glutamate Excitotoxicity
Downregulation of Postsynaptic Receptors
url https://doi.org/10.5281/zenodo.20040535