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| Main Author: | |
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| Format: | Recurso digital |
| Language: | English |
| Published: |
Zenodo
2026
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| Online Access: | https://doi.org/10.5281/zenodo.20095734 |
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Table of Contents:
- <p class="MsoNormal">Long-term diabetes mellitus and unstable glycemic control form a chain of reversible and irreversible changes in the renal parenchyma and microvascular system. Although diabetic nephropathy is clinically manifested by microalbuminuria and a decline in glomerular filtration rate, the pathogenesis centers on endothelial dysfunction, oxidative stress, low-grade inflammation, activation of the renin-angiotensin-aldosterone system, and tubulointerstitial fibrosis. Therefore, correcting renal damage not only at the symptomatic stage but also at the early stage, in a manner targeted at pathogenetic links, is one of the pressing tasks of modern nephrology and endocrinology. However, in practice, alongside pharmacotherapy, patients frequently resort to traditional medicine methods, which sharpens issues of clinical safety, drug interactions, and levels of evidence.</p>